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  1. Hi, i have a pharmacology question about SSRIs and in this instance lets take Zoloft as an example- Once you get past 100 mg of zoloft, lets say 150 mg don't you basically occupy SERT transporter as much as it will go therefore Is there really any subjective difference going up to 200? If so, how come? This can apply to all high dose SSRIs I'm confused by the principle of the matter
  2. Any robotic feelings i have are actually before i started risperdal and if its not from an endogenous disorder itself, i simply attribute it to my ssri but i have to take that, been on ssris for over a decade, if not i feel terrible even if i may have directly or indirectly more emotional sensitivity. Anyway i was hoping for some positivity from people saying their antipsychotic did not make them anhedonic
  3. Please be patient with me as part of why im asking is from my ocd, not to make any questioning of meds. I just want some positive reassurance. Just recently i had my risperdal increased to 3 mg to augment citalopram for ocd, depression. Thing is im nervous because i know it starts blocking a lot of dopamine. I know it cant be as simple as dopamine d2 equals pleasure and motivation? Im hoping the D1 receptors are more important and that the risperdal wont induce any worsening of anhedonic tendencies or amotivation in me. Surely its alpha 2 blockade may increase serotonin and norepinephrine. I guess im worried about two things. I hope i didnt just max out on the antidepressant properties of risperdal at 2 mg and now 3 is just a heavy dopamine blocker as hopefully its serotonin receptor occupancy will also increase dose dependently Plus im worried about all these things that implicate dopamine in reward and hope it isnt so clear cut that it will become problematic Perhaps anyone knowledgable can help elucidate my concerns or knowledge Thx and sorry about any trouble/ if my post is unclear
  4. i just recently hiked my risperdal up to 3 mg a day my understanding is that this is probably the max to augment ssri as 4-6 is usually used for psychosis. Not quite sure of pharmalogical rationale but risperdal does block alpha 2 adrenergic receptors in theory increasing serotonin and norepinephrine neurotransmission Maybe the d2 blockade will help ocd and will not induce anhedonia?
  5. Is gynecomastia risk with risperdal dose dependent? For example a male taking 2 mg for indications other than psychosis may not have the same risk as someone on 4 mg? I'm scared of this side effect, the last thing I want is to develop baseballs on my chest!
  6. It isn't for bipolar. It is merely to augment citalopram. Quite Frankly, I also worry if my decades of SSRI use has caused a permanent tolerance to their effectiveness. I dont even experience ssri sexual problems which i gather is rare. Is it really fine to be on antidepressants for decades? I guess my main hope is for risperidone to augment it to clear my mind
  7. I dont know why risperdal tho it does have studies. Im not sure it might help a lil or mentally i just feel reassured
  8. Hi, I take 40 mg of Citalopram everyday for years. Things are different than they used to be as far as SSRI effectiveness is concerned. I've been on SSRIs since i was a kid, im a young adult now, and they no longer work like they used to. I get obsessive thoughts, worries, not typical ocd rituals, and feel tense all the time, plus my mood dips I've been taking Risperdal for over a week now at 1 mg. Do you think its reasonable to ask my doctor if I can bump it up to 2 mg? For antidepressant and anti ocd effects, assuming dopamine blockade can aid some things How soon does risperdal work for nood
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